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" ... External to the spike protein, we again see many shared and unique mutations. In conjunction with D614G, a mutation to non-structural protein 12 (P323L) is widely dispersed among different variants. It likely enhances the capabilities of the polymerase, which is the mechanism that oversees viral replication. In NSP13, there are three unique mutations affecting the helicase, which aids in the replication process. These mutations likely also enhance this process, perhaps leading to increased viral load within the host body. ... "