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" ... More importantly, researchers identified the underlying mechanism causing the disease. Cells in the duodenum (the first part of the small intestine) no longer produced enough of an enzyme called intestinal alkaline phosphatase (IAP), which detoxifies the harmful lipopolysaccharide (endo)toxin produced by the Salmonella or other Gram-negative bacteria. This happened because the Salmonella induced another enzyme called Neuroaminidase (Neu), resulting in the IAP deficiency. Further, they found a potential treatment—feeding the mice with calf intestinal alkaline phosphatase (cIAP) resulted both in normalization of the levels and prevented the animals from developing symptoms. So did treating with Zanamivir, a (neuroaminidase inhibitor type) anti-viral used to treat influenza, sold as Relenza (NYSE:GSK). ... "